Relation of changes associated with inflamm atory diseases of parodontium and chronic obstructive pulmonary disease in smoking patients

Abstract

Frequency of interrelated changes in bronholegočnom and the mouth is from 54 % to 85 %. In this regard, the study of periodontal disease in the face of chronic obstructive pulmonary disease, so much so that in some cases, these diseases have similar etiology. The significance of combined periodontal pathology and bronchi is determined not only by the incidence and severity of diseases currents the negative influence on the organism as a whole, but also low treatment efficacy, especially against the backdrop of chronic tobacco smoking. načimyh pazličij or faktopnyh effects) took 0.05 functioning. Studies were conducted in 140 patients with inflammatory periodontal disease, the ones with COPD 107 patients (90 smokers, 17 non-smokers), COPD-patient 33 (26 smokers, 7 non-smokers). All patients voluntarily signed informed consent. The generally accepted definition was held dental indexes. To account for the number of microorganisms grown
on nutrient-dense Wednesday used counting colony forming units per 1 ml of physiological solution with suspendirovannym material of periodontal pockets, with recalculation of the biomass. Determine the forced expiratory volume in 1 second, the index Tiffno, vital capacity of the lungs, nicotine index index of the smoker. According to a study carried out by building a correlation as Pleiades structural diagrams. Statistical processing of material was performed using a standard software package for applied statistical analysis (Statistica for Windows v. 6.0). Critical zeroconfidence level statistical hypothesis (no significant difference or factor influences) were equal to 0.05. The reason for the wide dissemination of secondary edentulous patients with COPD smokers are more deeply destructive changes in periodontium amid tobacco smoking. Improving the dissemination of Streptococcus viridans Periodontal pockets corresponds to greater formation of plaque. Negative correlation of periodontal pockets of Streptococcus viridans contamination with LVC and positive with the number of plaque from smoking and non-smoking patients with COPD on the background of CDW in the absence of a statistically significant correlation in the groups of smokers and non-smokers
without COPD VZP patients may indicate involvement of Streptococcus viridans in the pathogenesis of COPD and EWR. Negative relationship lung capacity and forced expiratory volume for 1 second with index smoker testifies to the aggravating effects of tobacco smoking on the course of COPD.